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Interesting tid bit came in the mail

7 Aug

Information such as this below is just that for information – if you are terminally ill and is looking for anything that may help this can be one of those remedies you can try to help yourself.  Those of you who do not have a medical condition can test this out on yourself to see the benefit if there is any – always take this information with a grain of salt, however I can’t see anything negative happening by using orange peel.

 

DON’T WASTE THAT      LEMON PEEL/HEALTH BENEFITS: APPEARS      POSSIBLY 10,000 TIMES STRONGER THAN CHEMOTHERAPY

Many professionals in restaurants and eateries are using or  consuming the entire lemon and nothing is wasted.

How can you use the whole lemon without waste?

Simple..place the lemon in the freezer section of your refrigerator. Once the lemon is frozen, get your grater, and shred the      whole lemon (no need to peel it) and sprinkle it on top of your foods. Sprinkle it to your whisky, wine, vegetable salad, ice      cream, soup, noodles, spaghetti sauce, rice, sushi, fish dishes. All of      the foods will unexpectedly have a wonderful taste, something that you      may have never tasted before. Most likely, you only think of lemon juice and vitamin C.   Not anymore. Now that you’ve learned this lemon secret, you can use lemon  even in instant cup noodles.

What’s the major advantage of using the whole lemon other than preventing waste and adding new taste to your dishes? Well, you see lemon peels contain as much as 5 to 10 times      more vitamins than the lemon juice itself. And yes, that’s what you’ve been wasting. But from now on,      by following this simple procedure of freezing the whole lemon, then      grating it on top of your dishes, you can consume all of those nutrients      and get even healthier. It’s also good that lemon peels are health      rejuvenators in eradicating toxic elements in the body.

So place your lemon in your freezer, and then grate it on      your meal every day. It is a key to make your foods tastier and you get      to live healthier and longer! That’s the lemon secret! Better late than NEVER! The     surprising benefits of lemon! Lemon (Citrus) is a miraculous product to      kill cancer cells. It is 10,000 times stronger than chemotherapy.

Why do we not know about that?

Because there are laboratories interested in making a synthetic version that will bring them huge profits.

You can now help a friend in need by letting him/her know  that lemon juice is beneficial in preventing the disease. Its taste is pleasant and it does not produce the horrific effects of chemotherapy. How many people will die while this closely guarded secret      is kept, so as not to jeopardize the beneficial multimillionaires large      corporations?

As you know, the lemon tree is known for its varieties of      lemons and limes. You can eat the fruit in different ways: you can eat      the pulp, juice press, prepare drinks, sorbets, pastries, etc…

It is credited with many virtues, but the most interesting  is the effect it produces on cysts and tumours. This plant is a proven remedy against cancers of all types.   Some say it is very useful in all variants of cancer.

It is considered also as an anti microbial spectrum against  bacterial infections and fungi, effective against internal parasites and  worms, it regulates blood pressure which is too high and an      antidepressant,combats stress and nervous disorders.

The source of this information is fascinating: it comes from  one of the largest drug manufacturers in the world, says that after more  than 20 laboratory tests since 1970, the extracts revealed that it      destroys the malignant cells in 12 cancers, including colon, breast,    prostate, lung and pancreas …

The compounds of this tree showed 10,000 times better than the product Adriamycin, a drug normally used chemotherapeutic in the  world, slowing the growth of cancer cells.

And what is even more astonishing: this type of therapy with lemon extract only destroys malignant cancer cells and it does not  affect healthy cells.

 

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How can we remain healthy

26 Jun

Many of us look at Dr. Oz’s show and wish we could be like him. He is slim and appears to be in perfect health. He tells us to do the right things and we believe he takes great care of himself.  But doctors are people too and they admit to making lousy patients. Dr. Oz’s colon cancer scare must have been for scary for all of us who watch him. Because we ask ourselves what do you have to do to be healthy and to keep  deadly diseases at bay.  The truth is there is only so much one can do.  The truth is sometimes our fears bring on some of the illnesses we fear. We create what we focus on. If we focus on ill-health and avoiding illness we are putting our focus on the wrong thing. We have to focus on health and things that create health, refocus our thoughts to thinking about healthy and positively healthy scenarious. We come from a field of well-being. Read the article

http://www.huliq.com/10473/dr-oz-discusses-personal-colon-cancer-scare-time-magazine-f

Supermouse may help humans with cancer cure

28 Nov

supermouse.jpgScientists have developed a super Mice carrying a gene which appears to make them invulnerable to cancer. They are hoping that this  may hold the key to safer and more effective treatments for humans. The new breed, created with a more active “Par-4” gene, did not develop tumours, and even lived longer, said the journal Cancer Research. University of Kentucky researchers said a human cancer treatment was possible.

Excess Fat, Red Meat and Lack of Exercise Increase Cancer Risks

1 Nov

The official word is in The World Cancer Research Fund and the American Institute for Cancer Research has saidbadfood.jpg that excess body fat and red meat are linked to an increased risk of common cancers and should be avoided.  This has never been said before in such a definitive way so we had better start taking notice of this and follow the guidelines if we want to reduce our risk of escaping the claws of cancer.  This is not the first time consumers have been warned about red meat and excess body fat but now there is research to back it up in a 571 page comprehensive report released yesterday by 21 researchers. More than 7000 studies over five years were used in compiling the report. Researchers found that extra weight around the waist may lead to cancer of the esophagus, pancreas, colon, kidney and uterus as well as postmenopausal breast cancer.  The weight can be kept off  by exercising at least 30 minutes a day and limiting consumption of alcohol, high fat foods e.g. burgers, French fries, milk shakes, pastries and sugary drinks. Red means such as beef, pork and lamb are linked to colorectal cancers should be replaced with poultry fish and eggs.  Avoid also bacon and lunch meat.The Researchers found that about 40 percent of all cancers are linked to food, lack of exercise, and body weight. This can be used as a guide in designing cancer prevention education programs, and health policy around the world, panelists said.“Part of the purpose of the report was to show that prevention of cancer by means of food, nutrition, and associated factors is as feasible and crucial as prevention of coronary heart disease,” the researchers wrote. “The evidence that high body fatness and also physical inactivity are causes of a number of cancers, including common cancers, is particularly strong.”

Deadly Inheritance, Desperate Trade-Off

7 Aug

cance.jpg

When doctors found precancerous cysts, Nancy Platt chose to have her pancreas removed.

August 7, 2007

BALTIMORE — The room lights were dimmed, and all eyes turned to the grainy pictures on a pair of overhead monitors in a small room at Johns Hopkins Hospital.

Nancy Platt lay on her side, sedated, while Dr. Marcia Canto eased a long, camera-tipped tube down her throat to her stomach and began capturing ultrasound images of her pancreas.

Here and there, dark spots blinked onto the screen. They were pancreatic cysts — an ominous sign in someone like Mrs. Platt, 66, who lost her mother and a daughter to cancer of the pancreas. The cysts were unlikely to be cancerous, but given her family history, the odds were high that some would turn malignant.

An earlier test had also found cysts, and Mrs. Platt had traveled to Hopkins from her home near Houston to have part of her pancreas removed. Now, the picture was turning out to be even worse than expected.

“She has them all over the place,” said Dr. Canto, zeroing in on more cysts. Would she need to have her entire pancreas removed? asked a medical resident, eyeing the monitors. The pancreas makes insulin (and digestive enzymes), and removing it would cause severe diabetes that would require several insulin shots a day.

“It’s a big decision, but I don’t see how she can avoid it,” Dr. Canto said.

Mrs. Platt is part of a study aimed at preventing pancreatic cancer in people who are at high risk for it, by finding precancerous growths and removing all or part of the pancreas to get rid of them. So far, about 20 people have had the preventive surgery at Johns Hopkins, and a small number of others have undergone it at other centers.

In essence, these patients are trading the risk of cancer for the reality of diabetes, and their willingness to do it is a measure of the fear and desperation that pancreatic cancer provokes.

“With pancreatic cancer you don’t have much opportunity to save lives, and we are, with this approach,” said Dr. Canto, the director of endoscopy at Johns Hopkins.

Her program is strictly limited to high-risk families. Many other people have pancreatic cysts that turn up as incidental findings when they have scans for other reasons, and doctors are divided about whether to remove them.

Pancreatic cancer is one of the deadliest and most painful types of cancer. Even though it is only the 11th most common cancer in the United States, with 37,170 new cases expected this year, it is the fourth-leading cause of cancer death, because nearly all the patients die. This year, pancreatic cancer is expected to kill 33,370 people.

Ninety-five percent die in less than five years. Family history is a strong risk factor, but most patients have no affected relatives. Smoking, being over 60 and having diabetes or chronic pancreatitis increase the risk; so does being a man or an African-American.

It is not uncommon to hear of patients dying less than a year after the diagnosis. Mrs. Platt’s daughter, who was 37 and the mother of two young children, survived only three months.

The disease is so lethal partly because it is usually advanced by the time symptoms occur. The tumors cannot be felt during a physical exam because the pancreas lies deep in the abdomen, behind the stomach. And at every stage, the tumors resist chemotherapy and radiation.

“It is one of the most challenging human malignancies, for sure,” said Dr. Eileen O’Reilly, an oncologist who specializes in the disease at Memorial Sloan-Kettering Cancer Center in New York. “I honestly can’t think of a worse one.”

Even half-inch tumors in the pancreas are often fatal, while growths that size in the breast or intestine have a good prognosis, said Dr. Scott E. Kern, a molecular biologist at Johns Hopkins.

“Why? We don’t know,” Dr. Kern said, adding that even a small pancreas tumor “has all the mutations it needs to kill the patient.”

Scientists think that the many mutations, along with the tumors’ tendency to encase themselves in a capsule of scarlike tissue, help them resist chemotherapy. The organ’s natural ability to avoid being dissolved by its own digestive enzymes may also protect it from chemotherapy.

Researchers struggling to understand the disease are searching for genetic mutations that start cells down the path to cancer. They are also looking for biomarkers, molecules from abnormal cells that might be used for early detection, and they are studying the tumors themselves to find out what drives their growth, in hopes of blocking it. But many in the field say new ideas are badly needed.

Dr. Elizabeth M. Jaffee, who is trying to develop a vaccine against pancreatic cancer at Johns Hopkins, said, “I’m typically optimistic, but we are so far behind in this disease in understanding the biology, compared to breast or prostate cancer, that to expect therapies that will impact a good proportion of people who get this disease in the next few years, is very hard.”

Financing has lagged behind that for other cancers, and researchers say it is partly because there are not enough survivors to march on Washington.

Dr. Robert J. Mayer, the director of gastrointestinal oncology at the Dana-Farber Cancer Institute in Boston, said he thought research in pancreatic cancer would eventually catch up.

“When I started 30 years ago, people said you couldn’t treat advanced colon cancer, and now mortality is dropping in both genders, and the incidence is dropping with screening,” Dr. Mayer said. “I wouldn’t have believed it was possible. Nothing says we can’t do this in pancreatic cancer. It’s just more of a challenge.”

The first symptom of pancreatic cancer is often pain, because the tumors invade nerve clusters. Nausea, weight loss and jaundice may also occur.

“It’s a characteristic pain that goes almost to people’s backs,” Dr. Mayer said. “Years before CT scans, routine studies never found an explanation for that pain, and there was an apocryphal belief that the patients had depression, and depression led to pancreatic cancer. They would have weight loss and ill-defined pain and be sent to a psychiatrist, and later found to have pancreatic cancer. Now, this sort of pain syndrome leads to a CT scan.”

The only people who are cured or live more than a few years with this cancer have operable tumors — and not even all of them make it. About 15 percent to 20 percent of patients have surgery, and only about a quarter of them are cured. A recent report by the American College of Surgeons said that doctors are failing to offer the surgery to many patients who are candidates for it.

When the tumors are inoperable, it is often because they have invaded blood vessels and nerves. People who cannot be helped by surgery are usually given chemotherapy, which can slow the disease down for a while but not cure it.

The most common operation is called the Whipple procedure, and it is a long, difficult operation that usually involves removing parts of the pancreas, stomach, small intestine and other organs. Thirty years ago the operation itself had a death rate of 20 percent to 30 percent, and many doctors refused to recommend it even though it offered the only hope of a cure. But surgeons have improved the operation greatly over the years, and the mortality now is very low — in the right hands. Studies have shown that success rates are significantly higher, and complications lower, at hospitals that do many of these operations.

“I would go to a place that does this often,” Dr. Mayer said. “Compelling data indicate it, and reluctantly, I think, the general community has agreed. Ten or 15 places around the country have enormous experience, and I think it behooves one to go to a recognized cancer center. One might go to a perfectly fine community hospital for most other surgery, but I wouldn’t do it for this.”

Scientists are eager to study families that have been hard hit by pancreatic cancer. In 10 percent of patients, a parent or sibling also had the disease, and in 10 percent of those, multiple relatives have had it. In some, the disease is linked to a mutation in a gene called BRCA2, which can also increase the risk for breast cancer. Rare syndromes play a role in a few cases. But in most high-risk families, the gene is unknown.

“It’s such a deadly disease that it’s hard to find families with a lot of members alive,” said Dr. Ralph Hruban, the director of the pancreatic research center at Hopkins.

He said that finding the genes in high-risk families may also help researchers figure out the source of the more common cases, in which people have no affected relatives and apparently develop mutations later in life instead of being born with them.

“We hope to map genes related to familial pancreatic cancer,” said Dr. Gloria M. Petersen, an epidemiologist at the Mayo Clinic in Rochester, Minn. “The scientific underpinning is, you take the family tree, we know who has pancreatic cancer and who doesn’t, and everyone is genotyped.”

Then, the researchers look for markers — certain identifiable stretches of DNA — to see if any seem associated with the cancer. Finding one that tracks with the disease is the goal, Dr. Petersen said, because it helps researchers zero in on genes involved in the cancer. This approach is called a linkage analysis.

If a gene or genes is found, the most likely first use will be to help identify which family members really are at risk and in need of screening and, possibly, preventive surgery. That is what happened when high-risk genes for breast cancer and colon cancer were discovered.

Researchers always hope that finding a gene will lead them to a treatment that is able to kill or block the growth of cancer cells while leaving healthy cells unharmed. But only a few such “targeted” or “smart” drugs have been found.

Dr. Petersen was cautious about what her research might yield. “Is it going to lead to therapy or early detection?” she asked. “Look at the colon cancer and breast cancer story. They’re still struggling, in terms of a target for therapy. It depends on what the gene actually is and what it would do.”

Other research involves the tumor cells themselves. Cancers may have their own stem cells — a distinct, uniquely powerful subset of cells that cause most of a tumor’s growth and spread, and that seem invulnerable to chemotherapy and radiation. These cells could explain why some patients relapse even after treatment shrinks their tumors: drugs and radiation kill ordinary cancer cells, but the stem cells somehow survive and grow.

Studies have found evidence of these stem cells in blood, breast and brain cancers. In February, researchers at the University of Michigan reported in the journal Cancer Research that they had found the cells in tumors from people with pancreatic cancer. Implanted in mice, the stem cells were 100 times more likely than ordinary cancer cells to grow into tumors.

“We need to identify these small numbers of cells within a cancer, like we have,” said Dr. Diane M. Simeone, the senior author of the paper. “We need to better characterize them, and then we can figure out ways to selectively target or eradicate them. Then I think we’ll have much more effective therapies in hand. I don’t think it’s that far away.”

New drugs could be ready for testing in two or three years, she said.

“Numerous pharmaceutical companies are very interested in this, in working with investigators to come up with targeted therapies for these cells,” Dr. Simeone said. “I have at least a dozen banging on my door.”

Patients like Nancy Platt are poised on a razor’s edge between hope and despair. Their risk is high, but at least they know it and can try to do some thing about it.

“I don’t think I have any choice,” Mrs. Platt said, after learning that Dr. Canto had found multiple, scattered cysts. She decided to have her pancreas removed.

“The only thing that upsets me is that we couldn’t have done this for my daughter,” she said. But her surviving daughter, 36, has had an ultrasound test that found no abnormalities. Mrs. Platt’s son, 41, has yet to be tested.

Mrs. Platt had her pancreas removed in April. “There were lots and lots of cysts,” she said, adding she had no doubt that some would have become cancerous. “It was like a time bomb.”

Since then, guided by an endocrinologist, she has been learning to take care of her diabetes, which requires testing her blood at least four times a day, taking insulin shots before every meal and at bedtime, and using glucose pills to treat occasional attacks of low blood sugar brought on by the insulin.

But she is getting the hang of it, and is even planning a trip to Europe. She has no regrets.

She said, “They saved my life.”

Link between Cholesterol levels and Cancer

24 Jul

zLowering your cholesterol levels with statin drugs may slightly increase your risk of cancer, say U.S. researchers.Millions of North Americans take statin drugs to lower their cholesterol because it is widely considered one of the most important ways to prevent heart attacks and strokes.But an examination of the records of more than 41,000 patients in 23 different trials of statins, researchers found one extra case of cancer for every 1,000 patients with the lowest levels of low density lipoprotein (LDL), known more informally as bad cholesterol. The cancers were not of any specific type.Writing in the July 31 edition of the Journal of the American College of Cardiology, the authors stress that the benefits of statins still outweigh the risks.“The demonstrated benefits of statins in lowering the risk of heart disease remain clear; however, certain aspects of lowering LDL with statins remain controversial and merit further research,” Dr. Richard Karas of Tufts University School of Medicine in Boston said in a statement.The findings don’t directly implicate statins in increasing cancer risk, but they do raise important questions, said Karas, such as whether cancer risk is a possible side effect of statins or just low LDL. Statins such as Lipitor, Pravachol, Crestor, and Zocor lower LDL levels by blocking an enzyme in the liver responsible for making cholesterol.Previous anecdotal reports linked intensive LDL lowering with a higher incidence of health problems, including liver and muscle toxicity and cancer.The researchers concluded that moderate-dose therapy with multiple medications including statins may be better than high-dose therapy with statins alone.Dr. Karas emphasized that patients are advised to consult their doctor before discontinuing use of any medication.In an accompanying commentary published in the journal, Dr. John LaRosa of the State University of New York Downstate Medical Center in Brooklyn wrote that the research needs to be seen in perspective.“These current findings provide insufficient evidence that there is any problem with LDL lowering that outweighs its significant benefits on vascular disease,” he wrote.He suggested the higher cancer risk in patients with low LDL may be linked to the fact they live longer and are more likely to get cancer.

Summer and Sunscreen go Together

5 Jul

sunscreen.jpgJuly 5, 2007

Skin Deep

Do Sunscreens Have You

  

Covered?

AS the noon sun began to cook bathers in Long Beach, N.Y., last Sunday, members of the Sofferman family lounged on towels, each wearing a sun lotion chosen with the care usually given to picking out a new bathing suit.

Denise Sofferman and Ilene Sofferman, sisters who both work in the apparel industry in Manhattan, had put on tanning oil, their bodies already golden brown. Denise’s daughter, Lauren Levy, 21, a student at the University of Pennsylvania, had protected her pale skin with a heavy-duty S.P.F. 50 product formulated for children. Ilene’s 9-year-old daughter, Alison, had received a head-to-toe coating of S.P.F. 30.

Two hours later, the daughters were sunburned, their backs as pink as watermelon.

“It says waterproof, but Lauren didn’t even go swimming,” said Denise Sofferman, reapplying sunscreen to her daughter.

Ilene Sofferman, smearing another coat of lotion on Alison’s pink face, read from the back of the sunscreen bottle. “They have all these different marketing terms —S.P.F., UVA, UVB, waterproof, sweat-resistant — but you have to figure out what they mean by trial and error,” she said.

After decades of warnings about the dangers of sun exposure, an increasing number of Americans are making sunscreen part of their skin-care routines. Americans bought 60 million units of sunscreen last year, a 13 percent increase compared with 2005, according to Information Resources Inc., which tracks cosmetics sales.

But the increased demand has spurred an explosion of lotions, sprays, pads and gels with such diverse marketing claims — All-day Protection! Ultra Sweatproof! Total Block! Continuous Protection! Ultra Sport! Instant Protection! Extra UVA Protection! — that the Soffermans are not alone in their confusion over how to choose the most effective sunscreen.

In the nearly 30 years since the Food and Drug Administration issued its first regulations for sunscreen as an over-the-counter drug intended to reduce sunburn risk, the science surrounding skin and cancer has expanded dramatically.

Critics have clamored for the F.D.A to update the rules, saying that the standards have not kept pace. At the same time, they complain, the agency has allowed manufacturers to make vague and improbable-sounding marketing claims, leaving consumers confused and, worse, misled about what to use and how to use it to protect themselves.

The pressure on the agency has been mounting in recent weeks. Last month, reports by Consumer Reports and by the Environmental Working Group, a nonprofit group in Washington, found that a variety of popular sunscreens lacked sufficient broad protection against the sun’s harmful rays. And in May, Richard Blumenthal, Connecticut’s attorney general, sent a scathing petition to the F.D.A. saying that unclear sunscreen labels and inflated marketing put people at risk.

“Most sunscreens are deceptively and misleadingly labeled, most perniciously to give consumers a false sense of security,” Mr. Blumenthal said last week. “In my view, the F.D.A.’s failure to act is unconscionable and unjustifiable in any public sense.”

John Bailey, the executive vice president for science at the Cosmetics, Toiletry and Fragrance Association, an industry trade group, said that the directions on sunscreens adequately convey coverage. “These are very beneficial products which should be used to protect against the adverse effects of sunlight,” said Dr. Bailey, who has a Ph.D. in chemistry.

Nonetheless, the F.D.A. seems poised to address the labeling issue. Although it has been planning since 1999 to confirm new rules, Rita Chappelle, a spokeswoman for the F.D.A., said the agency expected to issue new sunscreen standards in the coming weeks. But until they are released, Ms. Chappelle said the agency would not answer questions about forthcoming regulations.

One fact about sunscreens is indisputable: They can impede sunburn and lower the incidence of at least one form of skin cancer in humans.

Dr. Allan C. Halpern, chief of dermatology at Memorial Sloan-Kettering Cancer Center in Manhattan, said that the regular use of sunscreen can inhibit squamous cell carcinoma, a cancer that kills 2,000 to 2,500 Americans a year.

In a study of about 1,600 residents of Nambour, Australia, volunteers who were given sunscreen to use every day for four and a half years had 40 percent fewer squamous cell cancers than a control group who maintained their normal skin-care routines. Even 10 years after the study concluded, the volunteers assigned to use sunscreen during the trial period had fewer cancers.

“It shows that using sun protection for almost five years gives you an intense, longer-term benefit against squamous cell carcinoma,” said Dr. Adèle C. Green, deputy director of the Queensland Institute of Medical Research in Brisbane, Australia, which ran the study.

Dr. Halpern said that sunscreen should also protect against melanoma, the deadliest skin cancer, and basal cell carcinoma, because the product can inhibit harmful ultraviolet rays that can contribute to the diseases.

Yet even after new F.D.A. labeling rules are published, it may take two years for the changes take effect.

Dr. James M. Spencer, a dermatologist in St. Petersburg, Fla., who specializes in skin cancer, said that he hopes the updated standards will clarify how much protection sunscreens provide, the dose needed to achieve significant protection, and the frequency with which a sunscreen should be reapplied.

The F.D.A. in 1978 first proposed a system of labeling products with an S.P.F. or Sun Protection Factor, which measures how effective the product is in preventing burn caused by the sun’s ultraviolet B rays. UVB radiation can also be a factor in skin cancer.

Dr. Spencer said that an S.P.F. 15 product screens about 94 percent of UVB rays while an S.P.F. 30 product screens 97 percent. Manufacturers determine the S.P.F. by dividing how many minutes it takes lab volunteers to burn wearing a thick layer of the product by the minutes they take to burn without the product.

But people rarely get the level of S.P.F. listed because labels do not explain how much to use, said Dr. Vincent A. DeLeo, chairman of dermatology at St. Luke’s-Roosevelt Hospital Center in Manhattan.

“Sunscreen is tested at 2 milligrams per square centimeter of skin, which means you should be using two ounces each time to cover your whole body,” Dr. DeLeo said. “But for most people an eight-ounce bottle lasts the whole summer.”

People who apply S.P.F. 30 too sparingly, for example, may end up with only S.P.F. 3 to S.P.F. 10, according to the Web site of the British Columbia Centre for Disease Control, www.bccdc.org/downloads/pdf/rps/reports/RIN15.pdf, which has comprehensive guidelines.

“The S.P.F. is a terrible system to guide consumers,” Dr. Spencer said. “Nobody is using sunscreen the way it is measured in a lab.” He said he hopes that the new standards will call for S.P.F. to be replaced with a system defining sun protection as high, medium or low.

Until then, Dr. Spencer said that people should use about a shot glass of sunscreen for the body and a teaspoon for the face to best achieve the S.P.F. protection listed on labels. It should be reapplied every few hours and immediately after swimming or sweating.

Dermatologists said that the agency is also likely to introduce a rating system for the sun’s ultraviolet A rays, which can contribute to cancer and skin aging. Many products already contain UVA screening agents, but under the current rules there is no rating for them.

Manufacturers are catching on that some consumers seek UVA protection. In print advertisements this month, Neutrogena and Banana Boat have been battling for UVA supremacy, including graphs in which each shows their product offering the highest coverage.

But Dr. David M. Pariser, the president-elect of the American Academy of Dermatology, said that without a standardized UVA rating system, consumers can’t be sure how much a sunscreen provides.

“Right now, we don’t know whether doubling the percentage of a UVA sunscreen ingredient doubles UVA protection or not,” Dr. Pariser said. “That is part of the muddled system we hope will be cleared up.”

Until then, Dr. Pariser said to choose sunscreens that contain ingredients known to filter UVA. These include Mexoryl SX, avobenzone, titanium dioxide and zinc oxide. He also recommended a database at www.cosmeticsdatabase.com/special/sunscreens/summary.php created by the Environmental Working Group that lists products with UVA protection.

Some doctors, along with Mr. Blumenthal of Connecticut, predicted that the new sunscreen rules would prohibit outsized marketing terms.

“ ‘All-day protection’ is just plain false since sunscreen has to be frequently reapplied,” Mr. Blumenthal said. “And ‘waterproof,’ which may be O.K. for an adult taking a quick dip in the pool but not for kids who are in and out of the water all day, is just plain deceptive.”

Dr. Green in Australia said the best way to prevent skin cancer is to stay out of the sun during peak hours and wear sun-protective clothing. But Dr. Halpern said you can’t keep Americans wrapped up.

“There is only a small subset of American society that is willing to wear long-sleeved shirts and wide-brimmed — defined as four inches wide — hats on a sunny day at the beach,” he said. “Until we can get that behavior, the next best thing is sunscreen. Put on two coats, so you won’t miss any spots.”

Precautions

Protection from the sun’s harmful rays requires more than slapping on a coating of cream. There are multiple precautions to take, according to interviews with doctors.

• Avoid outdoor activities during peak sun hours of 10 a.m. to 4 p.m.

• Wear protective clothing, sunglasses and wide-brimmed hats.

• If you are prone to burn, use a sunscreen with S.P.F. 30 or higher.

• Apply about a teaspoon of sunscreen to your face and a shot glass of it to your body.

• Make sure your sunscreen contains at least one ingredient known to filter UVA rays, such as zinc oxide, titanium dioxide, avobenzone or Mexoryl SX.

• Reapply sunscreen frequently, and immediately after swimming.

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